CHAPTER 1: A theory is put forward in which change in the
level of cellular energy charge in response to the
differing energy demands of the sleeping/waking
rhythm is the fundamental reason why sleep is
associated with restorative processes.
Numerous reports from the literature are
presented in which the time of sleep is associated
with a higher rate of synthesis.
CHAPTER 2: The literature is surveyed relating eating,
sleeping and body maintenance.
Hunger is associated with motor restlessness
and feeding with sedation.
Human studies indicate that a bedtime snack
of milk and cereal promotes sleep.
Losing weight leads to a reduction whereas
gaining weight leads to an increased amount of sleep.
CHAPTERS 3 and 4:
A milk and cereal food (Horlicks) had no effect
on sleep, whereas nitrazepam 5mg improved sleep.
Withdrawal from the drug led to disrupted sleep
A placebo pill had no effect on sleep.
Nitrazepam had no significant effect on the
plasma growth hormone, glucose, triglycerides or
cholesterol, but prolonged Horlicks administration
elevated triglyceride levels.
CHAPTERS 5 to 7: The effects on sleep were compared among a
placebo capsule, milk, Horlicks and a drink
nutritionally equivalent to Horlicks but containing
no milk or cereal. None of the three food drinks
had any significant effect on sleep when compared
with the inert capsule, but after Horlicks at bedtime
sleep was less broken than after the other two food
The dietary habits of subjects were found to
have a considerable influence on how they slept after
food at bedtime.
CHAPTERS 8 and 9: Correlational analysis revealed that body weight,
but not I.Q. was highly correlated with the mean
amount of REM sleep.
CHAPTER 10: It was also found that the mean sleep cycle length
correlated with the degree of over or under-weight.
CHAPTER 11: I attempt to answer questions raised by the
findings described in earlier chapters.
tentatively propose a theory linking the
maintenance of body weight, sleep cycle length and
amounts of REM sleep